Therapeutic vaccine candidates working with viral vectors encoding LMP2 have productively induced virus-unique CD8⁺ T mobile responses in early-phase clinical trials. Additionally, adoptive T cell therapies like posoleucel also goal LMP2 alongside other latency proteins for example EBNA1, reinforcing its importance for a conserved and immunodominant antigen.
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Amongst these latency proteins, EBNA-3a plays a supportive but non-critical role in B mobile transformation: EBV mutants missing EBNA-3a can still transform B cells, but with significantly decreased efficiency.
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